Vaskụla insufficiency

Vaskụla insufficiency n'akparamagwa banyere 1-2% nke ngụkọta bi. Ndị dị otú ahụ a na steeti bụ mgbe a N'ihi ya nke ọ bụla ọrịa. Na ọtụtụ mgbe, ọ na-eduga ná ọrịa nke obi usoro. Ndị a gụnyere cardiomyopathy, rịaworo, myocardial infarction, obi ntụpọ.

The mmeri nke obi muscle akpali mebiri nke inweta ọbara ahụ, n'ihi na ya na-agwụ na unsatisfactory arụmọrụ nke ya pumping ọrụ.

Vaskụla insufficiency e ji dị ka a enweghị nchịkwa ọnọdụ bụ nke a ọnụ na tonus nke ezigbo muscle arịa mgbidi. Nke a na-akpalite hypotension, nsogbu na venous laghachi na oruru ọbara.

Vaskụla insufficiency ruru ka isi ọnya ọdịdị nke obi odida esonyere ngwa na-apụghị izere ezere mmeghachi omume arịa. Nke a mmeghachi omume e ji a compensatory okike. N'ihi ya nnukwu cardio vaskụla insufficiency esonyere mmeghachi omume, nke na-egosipụta na ụdị vasoconstriction omume mmetụta nke pressor usoro. Nke a na-eduga onye na-abawanye na vaskụla eguzogide maka ụfọdụ oge, ụfọdụ na-abawanye na larịị nke ọbara mgbali na normalisation nke inweta ọbara mkpa akụkụ nke ndụ. Na-adịghị ala ala ọnọdụ dochie vasoconstriction abịa hypertrophy ire ụtọ muscle sel nke arịa mgbidi.

Na depletion nke compensatory usoro obi ọdịda, vaskụla insufficiency enyekwara. Nke a na-ebelata ngụkọta elu-eguzogide, n'ụzọ dị ịrịba ama ịgbasa obere veins, venules na capillaries.

Fọrọ nke nta ka ọ bụla usoro nke obi nwere na-arụ ọrụ strenuously maka a ogologo oge, ma ọ bụ bughi mmebi aa na myocardium, na-akpalite odida nke obi na arịa. Dị ka omume na-egosi na, mgbe mgbe, na steeti hụrụ na ndabere nke ọrịa ndị dị ka akwara akwara ọrịa, valvular ọrịa obi na ọrịa (enwetara na congenital), myocarditis, hypertensive ọnọdụ, cardiomyopathy. Ke adianade do, ọrịa nwere ike ime na ọrịa nke endocrine uwa metabolic ọnya, ezughi oke oriri na-edozi.

The kasị akpata ọnwụ (80% ikpe) n'oge ọrịa obi bụ akwara akwara ọrịa.

Iji ọrịa pụrụ iduga iche iche mere. Ọkachamara bụ atọ isi dị iche iche nke etiological ihe.

The mbụ gụnyere ihe na-nwere a kpọmkwem emebiri mmetụta na ndị myocardium. Ọ pụrụ ịbụ n'ụzọ a trauma, chemical ikpughe (eg, ọgwụ ovadozu). Ke adianade do, kpọmkwem emebiri mmetụta nwere ike, na ndu ihe (toxins, pathogens, nje).

Nke abụọ otu ihe mere na-agụnye ihe ndị na-akpasu bufee nke myocardial ọtọ agwa. Ndị a gụnyere ndị gabigara abawanye na olu nke na-abata ọbara ka obi ( "bufee ikike"). Nke a nwere ike ime mgbe gbasara obi valvụ insufficiency, ọnụnọ extracardiac na intracardiac shunts, na hypovolemia. bufee na myocardial akpata ntọt ke eguzogide n'elu ejection si uji eze nke obi ọbara ( "mgbali bufee"). Na nke a, obi odida amalite na ọnụnọ nke myocardial hypertrophy. Ọ ga-kwuru na hypertrophy emee ke idem ngalaba, nke na-eburu si ufiop ọrụ.

Na-atọ na-agụnye ihe ndị na-emetụta mkpọchị diastolic ventricular ndochi. Nke a ọnọdụ e mere site a ọnụ ruo n'ókè dị ukwuu na-ekesa ọbara olu (n'oge ike ujo ma ọ bụ ọbara na ọnwụ), na mebiri na diastolic ntụrụndụ nke obi n'oge mkpakọ nke ya mmiri mmiri nke accumulates na pericardium (ọbara, transudate, exudate).